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MyraCetill™ - Reduces the appearance of cellulite
Topical Use
Cellulite is caused because fat isn’t stored in a continuous layer under the skin. Fat is stored in fat cells (adipocytes) that are gathered in small bundles in between connective tissue that is made primarily of collagen produced by fibroblasts (cells manufacturing connective tissue which sustains healthy cellulite-free skin.)
When these “fat cell chambers” are burgeoning with fat, they protrude up into the dermal layer and create the lumpy texture that is seen as cellulite. Dihydromyricetin attacks cellulite by building-up collagen matrix and decrease in “fat cell chambers”.
Dihydromyricetin:
- inhibits 3 key proteins involved in the proliferation and differentiation of adipocytes. It inhibits adipogenesis (the creation of new fat cells) by inhibiting calveolin-1, a protein that is needed to make specialized adipocyte surface structures, GLUT-4, which allows glucose uptake in the cells, and perilipin, which
- promotes the selective inhibition of tyrosine kinase activity at the membrane receptor of the adipocyte.
- inhibits lipogenesis (the creation of more fat inside fat cells) by preventing fatty acids and sugars from entering adipocytes
- promotes lipolysis (fat burning inside adipocytes) by inhibiting proteins that protect fat globules, making the fat available for burning dermis, masking the fat cells underneath more effectively, and promoting a smoother skin surface
Dihydromyricetin has been shown to inhibit 3 key proteins involved in the proliferation and differentiation of adipocytes. Adipocytes have a remarkable surface full of calveoli, which are tiny caves acting as docks where fatty acids are transported into the fat cells. By inhibiting Calveolin and turned into fat molecules.
The higher the dermal density, the less cellulite is visible. Dihydromyricetin provided a +11.4% mean increase in dermal density during a human study of 10 subjects who applied twice daily a cream with 0.5% dihydromyricetin.
P.L. Thomas offers high-quality dihydromyricetin, extracted and purified by chromatography, specially formulated for cosmetic preparations.
Dihydromyricitin results in fewer fat cells; more fat-burning as measured by detecting the concentration of fat degradation compounds (glycerol); and less synthesis of fats within adipocytes. In each of these tests the result show the excellent concentration-dependent effects of dihydromyricitin offered by P.L. Thomas.
ResearchLiterature
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MyraCetill™ - Reduces the appearance of cellulite
Topical Use
Cellulite is caused because fat isn’t stored in a continuous layer under the skin. Fat is stored in fat cells (adipocytes) that are gathered in small bundles in between connective tissue that is made primarily of collagen produced by fibroblasts (cells manufacturing connective tissue which sustains healthy cellulite-free skin.)
When these “fat cell chambers” are burgeoning with fat, they protrude up into the dermal layer and create the lumpy texture that is seen as cellulite. Dihydromyricetin attacks cellulite by building-up collagen matrix and decrease in “fat cell chambers”.
Dihydromyricetin:
- inhibits 3 key proteins involved in the proliferation and differentiation of adipocytes. It inhibits adipogenesis (the creation of new fat cells) by inhibiting calveolin-1, a protein that is needed to make specialized adipocyte surface structures, GLUT-4, which allows glucose uptake in the cells, and perilipin, which
- promotes the selective inhibition of tyrosine kinase activity at the membrane receptor of the adipocyte.
- inhibits lipogenesis (the creation of more fat inside fat cells) by preventing fatty acids and sugars from entering adipocytes
- promotes lipolysis (fat burning inside adipocytes) by inhibiting proteins that protect fat globules, making the fat available for burning dermis, masking the fat cells underneath more effectively, and promoting a smoother skin surface
Dihydromyricetin has been shown to inhibit 3 key proteins involved in the proliferation and differentiation of adipocytes. Adipocytes have a remarkable surface full of calveoli, which are tiny caves acting as docks where fatty acids are transported into the fat cells. By inhibiting Calveolin and turned into fat molecules.
The higher the dermal density, the less cellulite is visible. Dihydromyricetin provided a +11.4% mean increase in dermal density during a human study of 10 subjects who applied twice daily a cream with 0.5% dihydromyricetin.
P.L. Thomas offers high-quality dihydromyricetin, extracted and purified by chromatography, specially formulated for cosmetic preparations.
Dihydromyricitin results in fewer fat cells; more fat-burning as measured by detecting the concentration of fat degradation compounds (glycerol); and less synthesis of fats within adipocytes. In each of these tests the result show the excellent concentration-dependent effects of dihydromyricitin offered by P.L. Thomas.
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